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CAS RN: 6402-23-9 | 产品编码: A0136
Acrinol Monohydrate
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技术规格
| Appearance | Light yellow to Yellow powder to crystal |
| Purity(HPLC) | min. 98.0 area% |
| Purity(Nonaqueous Titration) | min. 98.0 %(calcd.on anh.substance) |
| Solubility in hot Water | almost transparency |
| Water | 4.0 to 6.0 % |
物性（参考值）
| 熔点 | 245 °C |
| 水溶性 | 微溶 |
| 溶解性（微溶于） | 醇 |
GHS
| 象形图 |
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| 信号词 | Warning |
| 危险性说明 | H315 : Causes skin irritation. H319 : Causes serious eye irritation. |
| 防范说明 | P264 : Wash skin thoroughly after handling. P280 : Wear protective gloves/ eye protection/ face protection. P302 + P352 : IF ON SKIN: Wash with plenty of water. P337 + P313 : If eye irritation persists: Get medical advice/ attention. P305 + P351 + P338 : IF IN EYES: Rinse cautiously with water for several minutes. Remove contact lenses, if present and easy to do. Continue rinsing. P362 + P364 : Take off contaminated clothing and wash it before reuse. P332 + P313 : If skin irritation occurs: Get medical advice/ attention. |
相关法规
| RTECS# | OD4725000 |
运输信息
| HS编码* | 2933.99-000 |
应用
Ethacridine Lactate (another name: Acrinol): An Antibacterial Agent, an Inducer of Uterine Contractions and a PARG Inhibitor
Ethacridine lactate (other name: Acrinol or Ethodin) has been used as an antibacterial agent and as an inducer of uterine contractions.1,2) Recent studies have shown that ethacridine is an inhibitor of poly(ADP-ribose) glycohydrolase (PARG), which degrades poly(ADP-ribose) by hydrolyzing the ribose-ribose bonds present in poly(ADP-ribose).3,4) The inhibitors may induce apoptosis in cancer cells. (The product is for research use only.)
References
- 1) Antibacterial Properties of Ethacridine Lactate and Sulfmethoxazole Loaded Functionalized Graphene Oxide Nanocomposites
- 2) Ethodin: Pharmacological evidence of the interaction between smooth muscle and mast cells in the myometrium
- 3) Ethacridine Induces Apoptosis and Differentiation in Thyroid Cancer Cells In Vitro
- 4) Ibrutinib synergizes with poly(ADP-ribose) glycohydrolase inhibitors to induce cell death in AML cells via a BTK-independent mechanism.
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